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Arteriovenous Anastomoses and Cardiovascular Diseases
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Aim: Why are most of the cardiovascular diseases (CVD) with unknown etiology? An attempt to solve the riddle, An attempt to study the unknown role of the arteriovenous anastomoses (AVA), an attempt to finally clarify the causes of cardiac arrhythmia. Methods: Medical consultations, information search in literature, participation in medical conferences, correspondence with scientists, discussions with Russian leading cardiologists. Results: The official medicine: arteriovenous anastomosis (fistulas) - an abnormal connection between an artery and a vein. Normally, blood flows from arteries into the capillaries and then into the veins. Most cardiologists agree that the role of small or large AVA in human circulatory system is still poorly understood. We have found that along with the positive role, the anastomoses, especially large AVA, periodically have pathological effects on the cardiovascular system. The device “Cardiocode” was used for testing. Large arteriovenous anastomoses (AVA) can be opened under the influence of stress or physical loads. Periodically, the pressure change in the arteries and veins. Vena cava expands, its wall’s tone increases and pulse waves start to path through the AVA along the elastic walls of the vena cava to the right atrium and to the neck veins. Mechanical impulses can excite heart from various points of the atria or ventricles, disrupting the sinus rhythm. The result is the following: extrasystoles appear, tachycardia attacks, at the same time the blood flow is blocked on almost all the peripheral segments of circulatory system, edemata appear. Increased venous pressure stops the capillary circulation, which eventually leads to heart failure, even in a healthy heart. Severe metabolic disorders, it appears that leads to disease comorbidity, to venous congestion, to disease pelvic, to heart failure, to sudden cardiac death (SCD). Conclusions: I think we are approaching in the understanding of CVD. The absence of special breathing exercises, drinking large amounts of beer, smoking, lack of exercise and presence of large AVA can sometimes lead to a variety of diseases, to metabolic diseases. To get rid of the attacks of cardiac arrhythmias and the prevention of SCD we need to find some way to suppress the mechanical waves running through AVA, as “reentry” phenomenon has a mechanical nature. It is necessary to continue studying the AVA to develop new measures for neutralizing the pathological events associated with the open AVA.
Title: Arteriovenous Anastomoses and Cardiovascular Diseases
Description:
Aim: Why are most of the cardiovascular diseases (CVD) with unknown etiology? An attempt to solve the riddle, An attempt to study the unknown role of the arteriovenous anastomoses (AVA), an attempt to finally clarify the causes of cardiac arrhythmia.
Methods: Medical consultations, information search in literature, participation in medical conferences, correspondence with scientists, discussions with Russian leading cardiologists.
Results: The official medicine: arteriovenous anastomosis (fistulas) - an abnormal connection between an artery and a vein.
Normally, blood flows from arteries into the capillaries and then into the veins.
Most cardiologists agree that the role of small or large AVA in human circulatory system is still poorly understood.
We have found that along with the positive role, the anastomoses, especially large AVA, periodically have pathological effects on the cardiovascular system.
The device “Cardiocode” was used for testing.
Large arteriovenous anastomoses (AVA) can be opened under the influence of stress or physical loads.
Periodically, the pressure change in the arteries and veins.
Vena cava expands, its wall’s tone increases and pulse waves start to path through the AVA along the elastic walls of the vena cava to the right atrium and to the neck veins.
Mechanical impulses can excite heart from various points of the atria or ventricles, disrupting the sinus rhythm.
The result is the following: extrasystoles appear, tachycardia attacks, at the same time the blood flow is blocked on almost all the peripheral segments of circulatory system, edemata appear.
Increased venous pressure stops the capillary circulation, which eventually leads to heart failure, even in a healthy heart.
Severe metabolic disorders, it appears that leads to disease comorbidity, to venous congestion, to disease pelvic, to heart failure, to sudden cardiac death (SCD).
Conclusions: I think we are approaching in the understanding of CVD.
The absence of special breathing exercises, drinking large amounts of beer, smoking, lack of exercise and presence of large AVA can sometimes lead to a variety of diseases, to metabolic diseases.
To get rid of the attacks of cardiac arrhythmias and the prevention of SCD we need to find some way to suppress the mechanical waves running through AVA, as “reentry” phenomenon has a mechanical nature.
It is necessary to continue studying the AVA to develop new measures for neutralizing the pathological events associated with the open AVA.
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