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Learning from the worm: the effectiveness of protein-bound Moco to treat Moco deficiency
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Molybdenum cofactor (Moco) is synthesized endogenously in humans and is essential for human development. Supplementation of Moco or its precursors has been explored as a therapy to treat Moco-deficient patients but with significant limitations. By using the nematode C. elegans as a model, Warnhoff and colleagues (pp. 212–217) describe the beneficial impact of protein-bound Moco supplementation to treat Moco deficiency. If such an effect is conserved, this advance from basic research in worms may have significant clinical implications as a novel therapy for molybdenum cofactor deficiency.
Title: Learning from the worm: the effectiveness of protein-bound Moco to treat Moco deficiency
Description:
Molybdenum cofactor (Moco) is synthesized endogenously in humans and is essential for human development.
Supplementation of Moco or its precursors has been explored as a therapy to treat Moco-deficient patients but with significant limitations.
By using the nematode C.
elegans as a model, Warnhoff and colleagues (pp.
212–217) describe the beneficial impact of protein-bound Moco supplementation to treat Moco deficiency.
If such an effect is conserved, this advance from basic research in worms may have significant clinical implications as a novel therapy for molybdenum cofactor deficiency.
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