Search engine for discovering works of Art, research articles, and books related to Art and Culture
Javascript must be enabled to continue!
Supplementary Figures S1-S5 from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
View through CrossRef
<p>Supplementary Figures S1-S5. NPRL2/TUSC4 up-regulation doesn't correlate with patient survival (S1); Microarray analysis of NPRL2/TUSC4 knockdown cells (S2); The expression of NPRL2/TUSC4-deficient genes are correlated with canonical pathways changes (S3); Histogram of cell cycle distribution for control and NPRL2/TUSC4-deficiency cells (S4); Colony formation assay of NPRL2/TUSC4 overexpression and NPRL2/TUSC4 knockdown cells (S5).</p>
American Association for Cancer Research (AACR)
Title: Supplementary Figures S1-S5 from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
Description:
<p>Supplementary Figures S1-S5.
NPRL2/TUSC4 up-regulation doesn't correlate with patient survival (S1); Microarray analysis of NPRL2/TUSC4 knockdown cells (S2); The expression of NPRL2/TUSC4-deficient genes are correlated with canonical pathways changes (S3); Histogram of cell cycle distribution for control and NPRL2/TUSC4-deficiency cells (S4); Colony formation assay of NPRL2/TUSC4 overexpression and NPRL2/TUSC4 knockdown cells (S5).
</p>.
Related Results
Data from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
Data from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
<div>Abstract<p>BRCA1 expression is lost frequently in breast cancers in which it promotes malignant development. In the present study, we performed a global expression...
Data from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
Data from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
<div>Abstract<p>BRCA1 expression is lost frequently in breast cancers in which it promotes malignant development. In the present study, we performed a global expression...
TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
AbstractBRCA1 expression is lost frequently in breast cancers in which it promotes malignant development. In the present study, we performed a global expression analysis of breast ...
Abstract 1716: Evaluating the functional impacts of the BRCA1-mTORC2 interaction in breast cancer
Abstract 1716: Evaluating the functional impacts of the BRCA1-mTORC2 interaction in breast cancer
Abstract
Objective: The BRCA1 C-Terminal (BRCT) domain of BRCA1 has been found to interact with three accessory proteins (PRR5, RICTOR, and SIN1) of mTOR complex 2 (...
Supplementary Figures S1-S5 from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
Supplementary Figures S1-S5 from TUSC4 Functions as a Tumor Suppressor by Regulating BRCA1 Stability
<p>Supplementary Figures S1-S5. NPRL2/TUSC4 up-regulation doesn't correlate with patient survival (S1); Microarray analysis of NPRL2/TUSC4 knockdown cells (S2); The expressio...
Abstract 1716: BRCA1 expression modulates apoptosis and survival in response to retinoic acid in breast cancer cells
Abstract 1716: BRCA1 expression modulates apoptosis and survival in response to retinoic acid in breast cancer cells
Abstract
The vitamin A metabolite retinoic acid (RA) plays key roles in development, cell proliferation and differentiation. RA derivatives also display anti-cancino...
BRCA1 Expression Profile in Breast Cancer Patients in West Nusa Tenggara
BRCA1 Expression Profile in Breast Cancer Patients in West Nusa Tenggara
Abstract: Breast cancer is still the highest case in Indonesia. The mortality ratio: incidence of breast cancer in various countries can be influenced by differences in the clinica...
Abstract 2113: Elucidating mechanisms of regulation of homologous recombination utilizing a PALB2 fusion protein that contains the BRCT repeats of BRCA1
Abstract 2113: Elucidating mechanisms of regulation of homologous recombination utilizing a PALB2 fusion protein that contains the BRCT repeats of BRCA1
Abstract
The products of the breast cancer susceptibility genes, BRCA1 and PALB2, directly interact through coiled-coil domains present in each protein. How PALB2 is...