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Branch Retinal Artery Embolization Due to Calcific Aortic Valve Stenosis
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Purpose.
Branch retinal artery occlusion caused by calcific embolization secondary to calcific aortic valvulopathy
Methods.
A 45-year-old woman came to our attention complaining a sudden painless loss of her peripheral superior visual field. Best visual acuity was 20/20. Fundus examination revealed the presence of a retinal arterial embolic occlusion of the inferior branch. Fundus retinography visual field, and fluorescein angiography were performed and medical therapy was started. The echocardiography examination revealed a tricuspid and calcified aortic valve with moderate stenosis and regurgitation. Due to the heart pathology, the patient moved to the cardiosurgery department, where an aortic valve replacement was performed.
Results.
Four months after cardiac surgery, visual acuity of both eyes was stable (20/20). Fundus examination showed a complete reabsorption of the retinal edema and the resolution of retinal pallor. Fluorescein angiography confirmed the delay of the arterial filling. No retinal ischemia was observed. The visual field examination confirmed the deep scotoma previously registered.
Conclusions.
Retinal arterial embolization is a rare but potentially devastating complication of calcific aortic stenosis. Initial retinal presentation of calcific aortic stenosis is a rare condition. Keeping in mind that these emboli may be recurrent and potentially bilateral, a sudden onset of visual field defects, especially in young asymptomatic patients, needs immediate diagnosis and consideration of an urgent surgical correction.
Title: Branch Retinal Artery Embolization Due to Calcific Aortic Valve Stenosis
Description:
Purpose.
Branch retinal artery occlusion caused by calcific embolization secondary to calcific aortic valvulopathy
Methods.
A 45-year-old woman came to our attention complaining a sudden painless loss of her peripheral superior visual field.
Best visual acuity was 20/20.
Fundus examination revealed the presence of a retinal arterial embolic occlusion of the inferior branch.
Fundus retinography visual field, and fluorescein angiography were performed and medical therapy was started.
The echocardiography examination revealed a tricuspid and calcified aortic valve with moderate stenosis and regurgitation.
Due to the heart pathology, the patient moved to the cardiosurgery department, where an aortic valve replacement was performed.
Results.
Four months after cardiac surgery, visual acuity of both eyes was stable (20/20).
Fundus examination showed a complete reabsorption of the retinal edema and the resolution of retinal pallor.
Fluorescein angiography confirmed the delay of the arterial filling.
No retinal ischemia was observed.
The visual field examination confirmed the deep scotoma previously registered.
Conclusions.
Retinal arterial embolization is a rare but potentially devastating complication of calcific aortic stenosis.
Initial retinal presentation of calcific aortic stenosis is a rare condition.
Keeping in mind that these emboli may be recurrent and potentially bilateral, a sudden onset of visual field defects, especially in young asymptomatic patients, needs immediate diagnosis and consideration of an urgent surgical correction.
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