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Diagnosis and correction of venous dyscirculation in patients with cervicogenic headache

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Background. Cerebral venous circulation disorders occupy a significant place in the structure of vascular disorders in cervicogenic headache (CH). Available publications lack data on a comparative study of the state of venous cerebral hemodynamics in patients with CH during pathogenetic treatment of venous cerebral circulation disorders. Ruprose – dynamic study of cerebral venous blood flow and venous hemodynamic reactivity in patients with CH on the background of diosmin treatment. Materials and methods. A clinical and Doppler examination of 37 patients with CH aged 18 to 45 years was performed, in particular, cervicocranialgia (CCA) – 21 patients, posterior cervical sympathetic Barré–Lieu syndrome (BLS) – 16 patients. The maximum blood flow velocity (Vmax) in the vertebral (VV) veins, basal veins of Rosenthal (BV), straight sinus (SS), and the coefficient of reactivity to ortho­ (OL) and antiorthostatic (AOL) loading in the VV (CrvvOL, CrvvAOL) and SS (CrssOL, CrssAOL) as background indicators and against the backround of diosmin use were studied. Results. In patients with CH, Vmax was increased with an emphasis on BV in the CCA group and with an emphasis on VV and SS in the BLS group. In OL in VV in the groups of patients with CCA and BLS, hyporeactivity was noted, more pronounced in CCA. CrssOLwere not significantly change SS, more pronounced in patients with BLS. Against the background of diosmin use there was a decrease in Vmax in VV, BV and SS, as well as a decrease in CrdsAOL. Conclusions. In CH, signs of venous dyscirculation were noted, and they were more pronounced in BV in patients with CCA, as well as in VV and SS in patients with BLS. Hyperreactivity to AOL was also found in SS, which indicates subclinical intracranial venous hypertension. Under the influence of diosmin treatment, an improvement in venous hemodynamics was observed, most pronounced in VV, as well as stabilization of CrssOL indicators. The tendency to normalize the changed indicators of venous blood flow against the background of diosmin treatment demonstrates the significance of the velocity indicators of blood flow in the cerebral veins for determining the degree of venous dyscirculation and monitoring the effectiveness of treatment in patients with CH.
Title: Diagnosis and correction of venous dyscirculation in patients with cervicogenic headache
Description:
Background.
Cerebral venous circulation disorders occupy a significant place in the structure of vascular disorders in cervicogenic headache (CH).
Available publications lack data on a comparative study of the state of venous cerebral hemodynamics in patients with CH during pathogenetic treatment of venous cerebral circulation disorders.
Ruprose – dynamic study of cerebral venous blood flow and venous hemodynamic reactivity in patients with CH on the background of diosmin treatment.
Materials and methods.
A clinical and Doppler examination of 37 patients with CH aged 18 to 45 years was performed, in particular, cervicocranialgia (CCA) – 21 patients, posterior cervical sympathetic Barré–Lieu syndrome (BLS) – 16 patients.
The maximum blood flow velocity (Vmax) in the vertebral (VV) veins, basal veins of Rosenthal (BV), straight sinus (SS), and the coefficient of reactivity to ortho­ (OL) and antiorthostatic (AOL) loading in the VV (CrvvOL, CrvvAOL) and SS (CrssOL, CrssAOL) as background indicators and against the backround of diosmin use were studied.
Results.
In patients with CH, Vmax was increased with an emphasis on BV in the CCA group and with an emphasis on VV and SS in the BLS group.
In OL in VV in the groups of patients with CCA and BLS, hyporeactivity was noted, more pronounced in CCA.
CrssOLwere not significantly change SS, more pronounced in patients with BLS.
Against the background of diosmin use there was a decrease in Vmax in VV, BV and SS, as well as a decrease in CrdsAOL.
Conclusions.
In CH, signs of venous dyscirculation were noted, and they were more pronounced in BV in patients with CCA, as well as in VV and SS in patients with BLS.
Hyperreactivity to AOL was also found in SS, which indicates subclinical intracranial venous hypertension.
Under the influence of diosmin treatment, an improvement in venous hemodynamics was observed, most pronounced in VV, as well as stabilization of CrssOL indicators.
The tendency to normalize the changed indicators of venous blood flow against the background of diosmin treatment demonstrates the significance of the velocity indicators of blood flow in the cerebral veins for determining the degree of venous dyscirculation and monitoring the effectiveness of treatment in patients with CH.

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