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Comorbidity of peptic ulcer disease and osteoporosis in aspect of calcium regulatory system
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The comorbidity of various diseases requires the use of drugs that affect common etiological and pathogenetic aspects, which reduces the risk of side-effect.The Aim.Find out the state of calcium regulating system in the comorbidity of peptic ulcer disease (PUD) with osteoporosis (OP), and the effect of their shifts on the acid secretion and regional microcirculation.Materials and methods.We examined 10 patients with comorbid relapse PUD with OP and a group of patients corresponding to them by gender, age and location of the ulcer, only with a relapse of PUD. In all patients, except for clinical and endoscopic studies, the levels of parathyrin, calcitonin, calcium and phosphorus in the blood were studied, the secretory function of the stomach, densitometry was performed.Results.Relapse of ulcer was characterized by a increase in parathyroid hormone and calcium, a decrease in blood phosphorus. These changes were more pronounced in patients with a comorbidity of PUD with OP and were accompanied by an increase in acid secretion and regional microcirculation.Conclusion.The comorbidity of PUD with OP is accompanied by functional changes in calcium regulating system, which is one of the common pathogenetic mechanisms of these conditions and requires correction in their treatment.
Title: Comorbidity of peptic ulcer disease and osteoporosis in aspect of calcium regulatory system
Description:
The comorbidity of various diseases requires the use of drugs that affect common etiological and pathogenetic aspects, which reduces the risk of side-effect.
The Aim.
Find out the state of calcium regulating system in the comorbidity of peptic ulcer disease (PUD) with osteoporosis (OP), and the effect of their shifts on the acid secretion and regional microcirculation.
Materials and methods.
We examined 10 patients with comorbid relapse PUD with OP and a group of patients corresponding to them by gender, age and location of the ulcer, only with a relapse of PUD.
In all patients, except for clinical and endoscopic studies, the levels of parathyrin, calcitonin, calcium and phosphorus in the blood were studied, the secretory function of the stomach, densitometry was performed.
Results.
Relapse of ulcer was characterized by a increase in parathyroid hormone and calcium, a decrease in blood phosphorus.
These changes were more pronounced in patients with a comorbidity of PUD with OP and were accompanied by an increase in acid secretion and regional microcirculation.
Conclusion.
The comorbidity of PUD with OP is accompanied by functional changes in calcium regulating system, which is one of the common pathogenetic mechanisms of these conditions and requires correction in their treatment.
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