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Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
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Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
American Association for Cancer Research (AACR)
Title: Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
Description:
Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption.
Related Results
Data from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
Data from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
<div>Abstract<p>Functional suppression of spindle checkpoint protein activity results in apoptotic cell death arising from mitotic failure, including defective spindle ...
Data from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
Data from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
<div>Abstract<p>Functional suppression of spindle checkpoint protein activity results in apoptotic cell death arising from mitotic failure, including defective spindle ...
Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption
Supplementary Figures S1-S7 from p31<sup>comet</sup> Induces Cellular Senescence through p21 Accumulation and Mad2 Disruption...
Disturbed proteostasis is the key event that triggers cellular senescence through the regulation of chromatin organization and genome stability
Disturbed proteostasis is the key event that triggers cellular senescence through the regulation of chromatin organization and genome stability
AbstractMammalian cells undergo irreversible proliferation arrest when exposed to stresses, a phenomenon termed cellular senescence. Various types of stress induce cellular senesce...
Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
Abstract
Understanding factors subserving pituitary cell proliferation enables understanding mechanisms underlying uniquely benign pituitary tumors. Pituitary tumor-...
Data from Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
Data from Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
<div>Abstract<p>Understanding factors subserving pituitary cell proliferation enables understanding mechanisms underlying uniquely benign pituitary tumors. Pituitary tu...
Data from Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
Data from Senescence Mediates Pituitary Hypoplasia and Restrains Pituitary Tumor Growth
<div>Abstract<p>Understanding factors subserving pituitary cell proliferation enables understanding mechanisms underlying uniquely benign pituitary tumors. Pituitary tu...
Abstract 2842: 5-aza-2’-Deoxycytidine induces cellular senescence in non-small cell lung cancer
Abstract 2842: 5-aza-2’-Deoxycytidine induces cellular senescence in non-small cell lung cancer
Abstract
Background: Lung cancer is one of the refractory malignancies and the leading cause of cancer-related death worldwide. Although prognosis of lung cancer was...

